Previous studies have reported that CPs express sex hormone receptors, including estrogen and progesterone receptors. Although CPs are considered benign tumors, they display a relatively high recurrence rate that might compromise quality of life. The primary treatment for CPs is surgery but it is often unsuccessful. Identification of sufficiently split double potentials on the ablation line might represent an electrophysiological endpoint in these patients.Ĭraniopharyngiomas (CPs) are rare tumors of the sellar and suprasellar regions of embryonic origin. The use of mini-electrodes may help target slow pathway potentials during AVNRT ablation. No recurrence occurred during 1 year of follow-up. Following ablation, an interval separating double potentials in sinus rhythm ≥15% of baseline tachycardia cycle length was associated with non-inducibility in all patients ( p < 0.0001). At the end of the procedure, double potentials on the ablation line were identified in all patients, only on mini-electrode bipolar tracings. Slow pathway potentials could be identified in 77% of cases on mini-electrode bipolar tracings, versus 15% on conventional bipolar tracings ( p = 0.0009). Thirteen consecutive patients were included (85% female, median age 46 years). Mapping and ablation were performed using an irrigated catheter equipped with 3 insulated mini-electrodes on the distal tip. We prospectively included patients referred for AVNRT in our center. We sought to see whether a new ablation catheter equipped with mini-electrodes may facilitate the mapping of slow pathway potentials for AVNRT ablation. To view the papers in pdf format, click on the "PDF Full-text" link, and use the free Adobe Reader to open them.Ĭatheter ablation of the slow pathway in patients with atrioventricular nodal reentrant tachycardia (AVNRT) is mainly performed using anatomical landmarks. PDF is the official format for papers published in both, html and pdf forms.You may sign up for e-mail alerts to receive table of contents of newly released issues.Issues are regarded as officially published after their release is announced to the table of contents alert mailing list.Iron deficiency is associated with impaired exercise tolerance, increased heart failure hospitalisations, and mortality. Iron deficiency results in reduced organ function of the heart and skeletal muscles, anaemia, impaired immune responses, deranged metabolism, and kidney injury. Increased losses are mainly due to gastro-intestinal bleeding. Reduced absorption is thought to be due to intestinal oedema, increased intestinal pH, increased hepcidin levels, and intestinal hypoperfusion, among others. Reduced intake could be due to decreased absolute amounts of iron or low iron bioavailability. The causes can be divided into the reduced intake, decreased absorption, and increased loss of iron. A series of global mechanisms contribute to systemic iron deficiency in heart failure and its consequences.
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